Crigler–Najjar (type 1 and 2) syndrome:

  • Is due to deficiency of enzyme uridine diphosphate glucuronyltransferase(ugt).
  • Autosomal recessive disorder.
  • Both types present in the early neonatal period with a rapid rise in serum bilirubin to very high levels despite phototherapy.
  • Kernicterus can develop.
  • Type 1 is more severe.

Gilbert syndrome

  • Autosomal recessive condition in which UGT1A1 activity decreases mildly in hepatocytes, typically resulting in a benign unconjugated hyperbilirubinemia.
  • The likelihood of severe hyperbilirubinemia is increased if the infant also has G6PD deficiency.